Do submerged roots hinder orthodontic treatment or the use of implants?

INTRODUCTION: In clinical practice, submerged roots are found with high frequency, and their presence can change the planning of dental movements and implant placement. OBJECTIVES: To provide explanations of possible developments in the area involved, according to the evolutionary stage of the process, at the time of diagnosis. DISCUSSION: After atrophy of the periodontal ligament and epithelial remnants of Malassez, ankylosis of the bone with the submerged root occurs, and initiates a process of replacement resorption. Until this process reaches the most advanced stage, this area represents an increased "bone" density, and if some care is not taken, this can generate resorption problems in the tooth to be moved. Whereas implants can be placed, despite the presence of the submerged root, irrespective of the stage of evolution. CONCLUSION: It is natural for the onset of alveolodental ankylosis and tooth replacement resorption to occur in submerged roots, and its stage of evolution will be decisive in the approach to be adopted in clinical planning.

Canines and inflammatory external apical resorption in healthy maxillary lateral incisors due to occlusal trauma: when to detect the position of maxillary canines, to prevent it?

JUSTIFICATION: Canines represent corners in the dental arch, and are important features in facial esthetics, as they support the upper lip, wing of the nose, and influence the nasolabial fold and the appearance of facial aging. In the laterality movements, the canines guidance coordinate the opening and closing of the teeth, saving the TMJ from sudden movements. DISCUSSION: As a result of the lack of eruption or the inadequate positioning of the maxillary canine, the loss of the laterality guide may occur, which will then occur in the maxillary lateral incisor, inducing lesions of "occlusal trauma", such as inflammatory root resorption. Likewise, without well positioned canines, there may be premature aging and change in facial esthetics. CONCLUSION: In order to avoid problems with eruption and positioning of the maxillary canines, early diagnosis is made by analyzing their position and their relationship with the other teeth, and in the three-dimensional context of the maxilla, between 8-10 years of age. Preventive measures can create bone space and direction so that the maxillary canines can occupy their position in the dental arch.

Do submerged roots hinder orthodontic treatment or the use of implants?

ABSTRACT Introduction: In clinical practice, submerged roots are found with high frequency, and their presence can change the planning of dental movements and implant placement. Objectives: To provide explanations of possible developments in the area involved, according to the evolutionary stage of the process, at the time of diagnosis. Discussion: After atrophy of the periodontal ligament and epithelial remnants of Malassez, ankylosis of the bone with the submerged root occurs, and initiates a process of replacement resorption. Until this process reaches the most advanced stage, this area represents an increased "bone" density, and if some care is not taken, this can generate resorption problems in the tooth to be moved. Whereas implants can be placed, despite the presence of the submerged root, irrespective of the stage of evolution. Conclusion: It is natural for the onset of alveolodental ankylosis and tooth replacement resorption to occur in submerged roots, and its stage of evolution will be decisive in the approach to be adopted in clinical planning.

RESUMO Introdução: A frequência, na prática clínica, das raízes submersas é elevada, e sua presença pode modificar o planejamento de movimentações osteodentárias e da instalação de implantes. Objetivo: Apresentar as explicações das possíveis evoluções na área envolvida, de acordo com o estágio evolutivo do processo, no momento do diagnóstico. Discussão: Depois da atrofia do ligamento periodontal e dos restos epiteliais de Malassez, o osso anquilosa-se com a raiz submersa, e se inicia a reabsorção por substituição. Até se chegar ao estágio mais avançado, essa área representa uma densidade "óssea" aumentada, o que pode gerar problemas reabsortivos no dente a ser movimentado, caso alguns cuidados não sejam tomados. Por outro lado, os implantes podem ser aplicados, apesar da presença da raiz submersa, independentemente da fase evolutiva. Conclusão: É natural que, em raízes submersas, se instale a anquilose alveolodentária e a reabsorção dentária por substituição, e o seu estágio evolutivo será determinante na conduta a ser adotada no planejamento clínico.

The two extremes of physiological tooth resorption in primary tooth with or without the permanent successor tooth.

INTRODUCTION: Assessment of two radiographic images reveals two distinct, extreme situations of physiological tooth resorption, characteristic of primary teeth with or without permanent successor, due to partial anodontia. DISCUSSION: In all primary teeth, rhizolysis begins after the completion of formation, thanks to the apoptosis of their cells. When apoptosis induced by cementoblasts has denuded the root of these cells, the process of rhizolysis inevitably begins: This will be accelerated by mediators arising from the pericoronal follicle. When there is no permanent successor due to partial anodontia, rhizolysis occurs extremely slowly, and months later, without the epithelial rests of Malassez that were dead due to apoptosis, alveolodental ankylosis becomes established, and the tooth will gradually be replaced by bone, still within a physiological context. CONCLUSION: Rhizolysis and physiological tooth resorption may occur rapidly or slowly, early or late, and this depends on the presence of the permanent tooth, or its absence due to partial anodontia.

A contraindication to orthodontic and endodontic treatment: periapical cemento-osseous dysplasia.

INTRODUCTION: The dental pulp is completely normal in teeth with periapical cemento-osseous dysplasia. However, orthodontic and endodontic treatments are contraindicated in cases with this injury. OBJECTIVE: Present some biological, clinical and imaging reasons opposing these contraindications and questioning which are the real ones impediments and the reasons for the lack of research on the disease, analyzing cases submitted to orthopedic treatment under controlled and ethically approved conditions. CONCLUSION: The clinician can act safely based in available knowledge and aware of the possible consequences of orthodontic movement in teeth with periapical cemento-osseous dysplasia, as well as in the proper way of making a safe and definitive diagnosis.

A contraindication to orthodontic and endodontic treatment: periapical cemento-osseous dysplasia

ABSTRACT Introduction: The dental pulp is completely normal in teeth with periapical cemento-osseous dysplasia. However, orthodontic and endodontic treatments are contraindicated in cases with this injury. Objective: Present some biological, clinical and imaging reasons opposing these contraindications and questioning which are the real ones impediments and the reasons for the lack of research on the disease, analyzing cases submitted to orthopedic treatment under controlled and ethically approved conditions. Conclusion: The clinician can act safely based in available knowledge and aware of the possible consequences of orthodontic movement in teeth with periapical cemento-osseous dysplasia, as well as in the proper way of making a safe and definitive diagnosis.

RESUMO Introdução: A polpa dentária é completamente normal nos dentes com displasia cemento-óssea periapical. Porém, os tratamentos ortodônticos e endodônticos estão contraindicados nos casos com essa lesão. Objetivo: Apresentar algumas razões biológicas, clínicas e imagiológicas, contrapondo-se essas contraindicações e questionando quais seriam os reais impedimentos e os motivos da falta de pesquisa sobre a doença, analisando casuísticas submetidas ao tratamento ortodôntico sob condições controladas e eticamente aprovadas. Conclusão: O clínico pode agir de forma segura embasado no conhecimento disponível e consciente das possíveis consequências da movimentação ortodôntica nos dentes com displasia cemento-óssea periapical, bem como na forma adequada de se fazer o diagnóstico seguro e definitivo da doença.

Orthodontics and Endodontics: clinical decision-making.

Endodontically treated teeth may be moved, as endodontic treatment is not a contraindication for orthodontic treatment. Apical periodontal repair begins when the periapical or pulp lesion has completely resolved. This may happen immediately after treatment if the filling material causes little or no irritation of periapical tissues, and particularly if the material is fully contained within the canal. When it leaks, a foreign body granuloma forms and persists for some months or indefinitely, depending on the composition of the filling material. Materials containing calcium hydroxide with no resin components undergo phagocytosis and disappear in some months, as macrophages gradually remove them. Materials containing resins, silicone, ionomers, zinc oxide-eugenol, bioceramics or gutta-percha remain in the site and induce the formation of foreign body granulomas. Although this does not preclude tooth movement, patients should be followed up every three months using periapical images to control the position of the material in relation to the tooth apex. "Pseudo" overfilling may be avoided if permanent filling is delayed until the time when orthodontic treatment is completed.

Orthodontics and Endodontics: clinical decision-making

ABSTRACT Endodontically treated teeth may be moved, as endodontic treatment is not a contraindication for orthodontic treatment. Apical periodontal repair begins when the periapical or pulp lesion has completely resolved. This may happen immediately after treatment if the filling material causes little or no irritation of periapical tissues, and particularly if the material is fully contained within the canal. When it leaks, a foreign body granuloma forms and persists for some months or indefinitely, depending on the composition of the filling material. Materials containing calcium hydroxide with no resin components undergo phagocytosis and disappear in some months, as macrophages gradually remove them. Materials containing resins, silicone, ionomers, zinc oxide-eugenol, bioceramics or gutta-percha remain in the site and induce the formation of foreign body granulomas. Although this does not preclude tooth movement, patients should be followed up every three months using periapical images to control the position of the material in relation to the tooth apex. "Pseudo" overfilling may be avoided if permanent filling is delayed until the time when orthodontic treatment is completed.

RESUMO Os dentes tratados endodonticamente podem ser movimentados, e não representam contraindicação para o tratamento ortodôntico. O reparo periodontal apical se inicia a partir do momento em que a causa da lesão pulpar e/ou periapical é eliminada. Isso ocorre de maneira imediata se o material obturador for pouco ou nada agressivo aos tecidos periapicais, especialmente se estiver limitado ao canal. Quando extravasado, haverá a formação de granuloma do tipo corpo estranho por alguns meses ou permanentemente, dependendo da composição do material obturador. Os materiais à base de hidróxido de cálcio sem componentes resinosos são fagocitáveis e, em alguns meses, desaparecem do local, pois os macrófagos irão gradativamente removê-los. Os materiais à base de resina, silicone, ionômero, óxido de zinco e eugenol, biocerâmicos e guta-percha permanecerão no local, induzindo granulomas periapicais do tipo corpo estranho, o que não inviabiliza a movimentação dentária, mas devem ser acompanhados a cada três meses com imagens periapicais, para um controle de sua relação espacial com o ápice dentário. A "pseudossobreobturação" pode ser evitada se a obturação definitiva for postergada para quando o tratamento ortodôntico terminar.

Concrescence: can the teeth involved be moved or separated?

The atrophy of the periodontal ligament places the tooth very close to the bone or another tooth, as occurs in unerupted teeth. The absent interdental bone and the lack of functional periodontal stimulus may lead to the fusion of the appositional layers of cement between the roots of the teeth. Concrescence almost always occurs in the region of the maxillary molars. Asymptomatic, it should always be remembered when the proper response to orthodontic movement is not obtained, and there is no apparent explanation. When surgically extracting a tooth and there is resistance, insisting will not be the best strategy. Moving the teeth with concrescence is not convenient, as it requires very intense forces. Once separated, these teeth can be considered normal for movement. It is possible to separate two teeth presenting concrescence, but it depends on the extension of the area, the surgical access and, especially, the clinical convenience. The tooth to be extracted will be repaired with new cement deposited in the sectioned area. The simple separation with the maintenance of the proximity and the lack of function of one of the teeth will cause a new concrescence. After a period of 1 to 3 months, the separated teeth are biologically prepared to be moved. The most important detail in this separation of teeth presenting concrescence is that the diagnosis should be made in advance, and not at the time of the intervention.

Concrescence: can the teeth involved be moved or separated?

ABSTRACT The atrophy of the periodontal ligament places the tooth very close to the bone or another tooth, as occurs in unerupted teeth. The absent interdental bone and the lack of functional periodontal stimulus may lead to the fusion of the appositional layers of cement between the roots of the teeth. Concrescence almost always occurs in the region of the maxillary molars. Asymptomatic, it should always be remembered when the proper response to orthodontic movement is not obtained, and there is no apparent explanation. When surgically extracting a tooth and there is resistance, insisting will not be the best strategy. Moving the teeth with concrescence is not convenient, as it requires very intense forces. Once separated, these teeth can be considered normal for movement. It is possible to separate two teeth presenting concrescence, but it depends on the extension of the area, the surgical access and, especially, the clinical convenience. The tooth to be extracted will be repaired with new cement deposited in the sectioned area. The simple separation with the maintenance of the proximity and the lack of function of one of the teeth will cause a new concrescence. After a period of 1 to 3 months, the separated teeth are biologically prepared to be moved. The most important detail in this separation of teeth presenting concrescence is that the diagnosis should be made in advance, and not at the time of the intervention.

RESUMO A atrofia do ligamento periodontal coloca o dente muito próximo do osso e/ou de outro dente, como ocorre em dentes não irrompidos. O osso interdentário ausente e a falta de estímulo funcional periodontal podem levar à fusão das camadas aposicionais de cemento entre as raízes dos dentes. A concrescência ocorre quase sempre na região dos molares superiores. Assintomática, ela deve sempre ser lembrada quando não se obtém a resposta adequada a uma movimentação ortodôntica, sem uma explicação aparente. Cirurgicamente, quando se está extraindo o dente e ele oferece resistência, insistir não será a melhor estratégia. Movimentar os dentes com concrescência não é conveniente, pois requer forças muito intensas. Depois de separados, esses dentes podem ser considerados normais para a movimentação. É possível separar dois dentes em concrescência, mas depende da extensão da área, do acesso cirúrgico e, em especial, da conveniência clínica. O dente a ser extraído vai reparar-se, com novo cemento depositado na região seccionada. A simples separação, com manutenção da proximidade e da falta de função de um dos dentes, fará com que uma nova concrescência se estabeleça. Transcorrido o período de 1 a 3 meses, os dentes separados poderão, biologicamente, ser movimentados. O detalhe mais importante nessa separação de dentes com concrescência é que o diagnóstico deve ser feito com antecedência, e não no momento da intervenção.

Jaws can be referred to as narrow or hypoplastic, but the term "atresia" is inaccurate!

In order to lead to insights and discussion on proper use of Orthodontics and Pathology-related terminology, particularly in cases of smaller-than-usual maxilla and mandible - that is, anomalous ones -, this study compared the conceptual meaning of the term "atresia." It is considered improper when referring to maxilla and mandible with deficient growth compared to development that is satisfactory enough to reach normal size. To identify smaller maxilla and mandible, the most proper and accurate term is hypoplastic maxilla or mandible. This is because "atresia" stands for an anomaly related to lumen blockage in hollow organs, which is not the case for neither maxilla nor mandible. Hypoplastic maxilla or mandible can be properly and specifically referred to as micrognathia.

There is no pulp necrosis or calcific metamorphosis of pulp induced by orthodontic treatment: biological basis.

To biologically explain why the orthodontic treatment does not induce pulp necrosis and calcific metamorphosis of the pulp, this paper presents explanations based on pulp physiology, microscopy and pathology, and especially the cell and tissue phenomena that characterize the induced tooth movement. The final reflections are as follows: 1) the orthodontic movement does not induce pulp necrosis or calcific metamorphosis of the pulp; 2) there is no literature or experimental and clinical models to demonstrate or minimally evidence pulp alterations induced by orthodontic movement; 3) when pulp necrosis or calcific metamorphosis of the pulp is diagnosed during orthodontic treatment or soon after removal of orthodontic appliances, its etiology should be assigned to concussion dental trauma, rather than to orthodontic treatment; 4) the two pulp disorders that cause tooth discoloration in apparently healthy teeth are the aseptic pulp necrosis and calcific metamorphosis of the pulp, both only induced by dental trauma; 5) the concussion dental trauma still requires many clinical and laboratory studies with pertinent experimental models, to increasingly explain its effects on the periodontal and pulp tissues.

Jaws can be referred to as narrow or hypoplastic, but the term "atresia" is inaccurate!

ABSTRACT In order to lead to insights and discussion on proper use of Orthodontics and Pathology-related terminology, particularly in cases of smaller-than-usual maxilla and mandible - that is, anomalous ones -, this study compared the conceptual meaning of the term "atresia." It is considered improper when referring to maxilla and mandible with deficient growth compared to development that is satisfactory enough to reach normal size. To identify smaller maxilla and mandible, the most proper and accurate term is hypoplastic maxilla or mandible. This is because "atresia" stands for an anomaly related to lumen blockage in hollow organs, which is not the case for neither maxilla nor mandible. Hypoplastic maxilla or mandible can be properly and specifically referred to as micrognathia.

RESUMO Para induzir reflexões e discussões sobre o uso adequado da nomenclatura em Ortodontia e Patologia, para os casos em que a maxila e a mandíbula apresentam-se pequenas ou menores do que o habitual, ou seja, anômalas, comparou-se o significado conceitual do termo "atrésica". Esse termo não é adequado quando aplicado à maxila e à mandíbula para identificar situações em que houve um desenvolvimento com crescimento insuficiente para se chegar ao tamanho normal. Para identificar maxila e mandíbula menores, é mais adequado e preciso o uso do termo maxila ou mandíbula hipoplásica. Isso porque atresia representa uma anomalia por obstrução da luz ou lume em órgãos ocos, o que não ocorre na maxila ou na mandíbula. Maxila ou mandíbula hipoplásica também podem ser chamadas, apropriada e especificamente, de micrognatia.

There is no pulp necrosis or calcific metamorphosis of pulp induced by orthodontic treatment: biological basis

ABSTRACT To biologically explain why the orthodontic treatment does not induce pulp necrosis and calcific metamorphosis of the pulp, this paper presents explanations based on pulp physiology, microscopy and pathology, and especially the cell and tissue phenomena that characterize the induced tooth movement. The final reflections are as follows: 1) the orthodontic movement does not induce pulp necrosis or calcific metamorphosis of the pulp; 2) there is no literature or experimental and clinical models to demonstrate or minimally evidence pulp alterations induced by orthodontic movement; 3) when pulp necrosis or calcific metamorphosis of the pulp is diagnosed during orthodontic treatment or soon after removal of orthodontic appliances, its etiology should be assigned to concussion dental trauma, rather than to orthodontic treatment; 4) the two pulp disorders that cause tooth discoloration in apparently healthy teeth are the aseptic pulp necrosis and calcific metamorphosis of the pulp, both only induced by dental trauma; 5) the concussion dental trauma still requires many clinical and laboratory studies with pertinent experimental models, to increasingly explain its effects on the periodontal and pulp tissues.

RESUMO Para fundamentar biologicamente por que o tratamento ortodôntico não induz necrose pulpar e metamorfose cálcica da polpa, apresentou-se explicações com base na fisiologia, microscopia e patologia pulpar, bem como, e principalmente, nos fenômenos celulares e teciduais que caracterizam a movimentação dentária induzida. As reflexões finais foram: 1) o movimento ortodôntico não induz necrose pulpar ou metamorfose cálcica da polpa; 2) não há literatura e modelos experimentais e clínicos que comprovem ou minimamente evidenciem alterações pulpares induzidas pelo movimento ortodôntico; 3) quando a necrose pulpar ou metamorfose cálcica da polpa for diagnosticada durante o tratamento ortodôntico ou logo após a remoção dos aparelhos ortodônticos, a sua etiologia deve ser atribuída ao traumatismo dentário do tipo concussão, e não ao tratamento ortodôntico; 4) as duas doenças pulpares que levam ao escurecimento coronário em dentes aparentemente hígidos são a necrose pulpar asséptica e a metamorfose cálcica da polpa, ambas induzidas exclusivamente pelo traumatismo dentário; 5) o traumatismo dentário do tipo concussão requer, ainda, muitos estudos clínicos e laboratoriais, com modelos experimentais pertinentes, para fundamentar cada vez mais os seus efeitos sobre os tecidos periodontais e pulpares.

"Maxillary lateral incisor partial anodontia sequence": a clinical entity with epigenetic origin

ABSTRACT The relationship between maxillary lateral incisor anodontia and the palatal displacement of unerupted maxillary canines cannot be considered as a multiple tooth abnormality with defined genetic etiology in order to be regarded as a "syndrome". Neither were the involved genes identified and located in the human genome, nor was it presumed on which chromosome the responsible gene would be located. The palatal maxillary canine displacement in cases of partial anodontia of the maxillary lateral incisor is potentially associated with environmental changes caused by its absence in its place of formation and eruption, which would characterize an epigenetic etiology. The lack of the maxillary lateral incisor in the canine region means removing one of the reference guides for the eruptive trajectory of the maxillary canine, which would therefore, not erupt and /or impact on the palate. Consequently, and in sequence, it would lead to malocclusion, maxillary atresia, transposition, prolonged retention of the deciduous canine and resorption in the neighboring teeth. Thus, we can say that we are dealing with a set of anomalies and multiple sequential changes known as sequential development anomalies or, simply, sequence. Once the epigenetics and sequential condition is accepted for this clinical picture, it could be called "Maxillary Lateral Incisor Partial Anodontia Sequence."

RESUMO A relação entre a anodontia parcial do incisivo lateral e o deslocamento palatino do canino superior não irrompido não pode ser considerada uma anomalia dentária múltipla com etiopatogenia genética definida, a ponto de ser considerada como uma "síndrome". Os genes envolvidos sequer foram identificados e localizados no genoma humano, e nem mesmo presumiu-se em qual cromossomo se localizaria o gene responsável. O deslocamento palatino do canino superior em casos de anodontia parcial do incisivo lateral superior está potencialmente associado às mudanças ambientais provocadas pela sua ausência no local de formação e erupção, o que caracterizaria uma etiologia epigenética para essa associação. A falta do incisivo lateral superior na região canina implica em tirar um dos guias referenciais da trajetória eruptiva do canino superior, que ficaria, assim, não irrompido e/ou impactado no palato. Como consequência, e em sequência, promove-se uma má oclusão, atresia maxilar, transposição, retenção prolongada do canino decíduo e reabsorções nos dentes vizinhos. Dessa forma, pode-se afirmar que estamos frente a um conjunto de anomalias e alterações múltiplas sequenciais conhecido como anomalias de desenvolvimento sequencial ou, simplesmente, sequência. Uma vez aceita a condição epigenética e sequencial para esse quadro clínico, ele poderia ser chamado de "Sequência da Anodontia Parcial do Incisivo Lateral Superior".

Bone Healing Evaluation in Critical-Size Defects Treated With Xenogenous Bone Plus Porcine Collagen.

PURPOSE: This study aimed to evaluate the osteoconductive features of Bio-Oss Collagen in large critical-size defects (8 mm). MATERIALS AND METHODS: Thirty-six adult Wistar Albinus male adult rats were carried out a critical-size defect with a trephine bur of 8 mm in the calvaria. Groups were divided depending on the filling biomaterial as follows: group BO: Bio-Oss (n = 18); group BOC: Bio-Oss Collagen (n = 18). After 7, 30, and 60 days, 6 rats of each group were euthanized with anesthetic overdose. Specimens were laboratory processed for histomorphometric analysis. Histomorphometric data were statistically analyzed by analysis of variance and post-Tukey test (P < 0.05). RESULTS: Statistical differences were found in new bone formation just in the intragroup comparison among periods after 7 and 60 postoperative days, indicating more new bone formation after 60 days (Tukey test, P = 0.029). CONCLUSION: Under the limitation of this research, it could be concluded that Bio-Oss and Bio-Oss Collagen in this experimental model did not show osteoconductive features.

"Maxillary lateral incisor partial anodontia sequence": a clinical entity with epigenetic origin.

The relationship between maxillary lateral incisor anodontia and the palatal displacement of unerupted maxillary canines cannot be considered as a multiple tooth abnormality with defined genetic etiology in order to be regarded as a "syndrome". Neither were the involved genes identified and located in the human genome, nor was it presumed on which chromosome the responsible gene would be located. The palatal maxillary canine displacement in cases of partial anodontia of the maxillary lateral incisor is potentially associated with environmental changes caused by its absence in its place of formation and eruption, which would characterize an epigenetic etiology. The lack of the maxillary lateral incisor in the canine region means removing one of the reference guides for the eruptive trajectory of the maxillary canine, which would therefore, not erupt and /or impact on the palate. Consequently, and in sequence, it would lead to malocclusion, maxillary atresia, transposition, prolonged retention of the deciduous canine and resorption in the neighboring teeth. Thus, we can say that we are dealing with a set of anomalies and multiple sequential changes known as sequential development anomalies or, simply, sequence. Once the epigenetics and sequential condition is accepted for this clinical picture, it could be called "Maxillary Lateral Incisor Partial Anodontia Sequence."

Dentes com vitalidade pulpar e lesão periapical: displasia cemento-óssea periapical / Teeth with pulp vitality and apical periodontitis: periapical cemento-osseous dysplasia

Apesar da afirmação dogmática de que "se o dente tem lesão periapical, não existe mais vitalidade pulpar", isso não corresponde à realidade, pois existem periapicopatias em que os dentes portadores permanecem com vitalidade pulpar. Para fundamentar esse raciocínio, será discutido no presente artigo: 1) O conceito de periapicopatia verdadeira, 2) As diferenças entre ápice e periápice, 3) A distinção entre pericemento apical e periodonto apical, e 4) Considerações sobre a classificação das periapicopatias verdadeiras. Entre as quatro situações clínicas em que o dente com lesão periapical mantém a vitalidade pulpar, escolheu-se, para este primeiro trabalho de uma série, a Displasia Cemento-Óssea Periapical, que não requer qualquer tratamento, apenas controle ao longo do tempo.

A origem dos toros palatinos e mandibulares: bases para a sua interpretação clínica / Origin of torus palatinus and torus mandibularis: basis for clinical interpretation

Os toros palatinos e mandibulares são distúrbios do desenvolvimento do tipo anomalia de forma, com manifestação tardia no crescimento e maturação dos maxilares. Os casos familiares e a persistência dos toros com a idade e em desdentados lhe atribuem uma origem genética e dificultam a sua interpretação como uma resposta adaptativa à sobrecarga oclusal, ao bruxismo e outros fatores externos: os toros não são hiperplasias ou hipertrofias adaptativas. Os toros são protuberâncias ósseas sem cápsula fibrosa, o que os diferencia dos osteomas e lhes tira a natureza neoplásica, mesmo que benigna, especialmente porque também não apresentam crescimento contínuo e sem controle por parte do organismo. O tamanho dos toros se estabiliza no final do crescimento dos maxilares, ao redor dos 22 a 24 anos. Os toros são constituídos de osso normal, do ponto de vista funcional e estrutural, e podem ser utilizados como sítio de origem de transplante ósseo autógeno para outros locais, ou como sede de implantes osseointegráveis, se houver conveniências clínicas para tais procedimentos...

Torus palatinus and torus mandibularis are developmental anomalies of shape that become late manifest during growth and maturation of the jaws. Family history and the constant presence of tori with age and among edentulous patients attach a genetic origin to them and hinder their clinical interpretation as an adaptive response to occlusal overload, grinding and other external factors: tori are not a form of hyperplasia or adaptive hypertrophy. They are bone protuberances without a fibrous capsule, which differentiates them from osteomata and frees them from a neoplastic nature, albeit benign, especially because they do not grow continuously and uncontrollably in one’s organism. The size of tori stabilizes by the end of maxillary growth, at around the age of 22 to 24 years. They are composed of normal bone, from a functional and structural perspective, and might be used as autograft harvesting site or osseointegrated implant placement site within clinically acceptable conditions...

Clarifications, guidelines and questions about the dental bleaching "associated" with orthodontic treatment.

With regard to the best moment for carrying out or recommending dental bleaching to orthodontic patients, some explanations and orientations are given in order to answers the following questions: 1) Why orthodontic treatment completion is considered the best opportunity for carrying out the procedure? 2) Why dental bleaching should not be performed immediately before orthodontic treatment? 3) If that would be possible at any special case, what would that be? 4) Why dental bleaching should not be performed during orthodontic treatment? 5) If that would be possible at any special case, what would that be? This article highlights why it is essential to protect both the mucosa and the cervical region, regardless of the moment when dental bleaching is performed, whether associated with orthodontic treatment or not. The "how", "why" and "if" it is or not convenient to perform dental bleaching before orthodontic treatment are still a matter of clinical suggestion, as it is a procedure that is under analysis, empirical knowledge waiting for scientific proof or disproof! Although tooth enamel has adamantine fluid flowing within it, providing a specific metabolism that is peculiar to its own and which could scientifically explain and base the option of carrying out teeth whitening before and during orthodontic treatment, we must still be very careful.